Symptoms of acute nephritic syndrome are similar to those of chronic nephritic disease, but there are some differences. Acute nephritic syndrome may present with a variety of different symptoms, including fluid accumulation in the body (edema), decreased urine volume (oliguria), and blood in the urine (hematuria). The latter is the most concerning symptom, as it can be classified into two different types of hematuria: microhematuria) is undetectable and macrohematuria is visible blood.
Dialysis is a process in which a machine mimics a kidney. It may be temporary or permanent, depending on the degree of damage to the kidneys. Treatment for acute nephritis generally involves bed rest and supportive measures, and evaluation tests may be necessary to diagnose and treat the underlying conditions. Patients must be closely monitored to monitor the disease’s progression and determine whether other treatments are needed.
Acute nephritic syndrome is an inflammatory disorder affecting the glomerulus, which filters blood to produce urine. This condition damages the kidney’s ability to remove waste and excess fluid. The body builds up excess fluid, which results in swelling. The kidneys cannot filter the blood, and blood in the urine is a sign of a more serious condition. If left untreated, acute nephritic syndrome can lead to kidney failure.
Among the many causes of acute nephritic syndrome, the most common is inflammation of blood vessels in the kidney. Inflammation in the blood vessels may also cause the kidneys to leak protein. Those who have it should be especially careful about their lifestyle, as it can lead to irreversible damage. In addition to kidney damage, acute nephritic syndrome may be caused by some drugs. Gold salts, for example, can impair the kidney’s ability to filter blood. Captopril, for example, is a drug that may worsen proteinuria.